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The aim of our studies is to provide better and personalized therapeutic modalities in breast cancer. Our approach is based on understanding the way the inflammatory/immune microenvironment affects pro-malignancy functions in breast tumor cells and its impact on tumor-stroma interactions.

How do inflammatory mediators drive metastatic traits in breast tumor cells?

Can we use inflammatory chemokines and immune molecules for drug design in breast cancer?

How does the inflammatory microenvironment impact tumor-promoting stromal cells?